Voluntary and forced exposure to ethanol vapor produces similar

A major limitation of the most widely used current animal models of alcohol dependence is that they use forced exposure to ethanol including ethanol-c…

Chronic ethanol alters adrenergic receptor gene expression and produces cognitive deficits in male mice - ScienceDirect

Frontiers Adolescent Alcohol and Stress Exposure Rewires Key Cortical Neurocircuitry

Voluntary and forced exposure to ethanol vapor produces similar escalation of alcohol drinking but differential recruitment of brain regions related to stress, habit, and reward in male rats - ScienceDirect

CIE and EVSA alcohol rats show similar BALs and level of escalation and

Genome-wide transcriptomics of the amygdala reveals similar oligodendrocyte-related responses to acute and chronic alcohol drinking in female mice

Alcohol withdrawal activated CRF neurons in the CeA. a Exposure to

Catechol-O-Methyltransferase inhibition and alcohol use disorder: Evaluating the efficacy of tolcapone in ethanol-dependent rats - ScienceDirect

PDF] Chronic intermittent voluntary alcohol drinking induces hyperalgesia in Sprague-Dawley rats.

Knockdown of BDNF expression within the DMS does not alter ethanol

Frontiers Linking Ethanol-Addictive Behaviors With Brain Catecholamines: Release Pattern Matters

Frontiers Moderate ethanol exposure during early ontogeny of the rat alters respiratory plasticity, ultrasonic distress vocalizations, increases brain catalase activity, and acetaldehyde-mediated ethanol intake

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Acute ethanol decreases excitatory synaptic transmission in CeA

Translational opportunities in animal and human models to study alcohol use disorder

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