Sodium valproate and 5-aza-2′-deoxycytidine differentially modulate DNA demethylation in G1 phase-arrested and proliferative HeLa cells
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Last updated 25 outubro 2024
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Mechanism of Sodium Valproate Combined with Decitabine in Inhibiting Hepatocellular Carcinoma Cells Epithelial-Mesenchymal Transition and Invasion and Metastasis Based on p38 Mitogen-Activated Protein Kinases/Heat Shock Protein 27 Regulating
Valproate-Induced Epigenetic Upregulation of Hypothalamic Fto Expression Potentially Linked with Weight Gain
DNA methylation status. The 5.8S and H3 histone genes, which lack HpaII
TET protein inhibitors: Potential and limitations - ScienceDirect
Combination treatment with DAC and Pano enhanced reactivation of
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PDF) Sodium valproate and 5-aza-2′-deoxycytidine differentially modulate DNA demethylation in G1 phase-arrested and proliferative HeLa cells
Exposure to hypomethylating 5-aza-2′-deoxycytidine (decitabine) causes rapid, severe DNA damage, telomere elongation and mitotic dysfunction in human WIL2-NS cells - ScienceDirect
Transcriptome and methylome changes in U937 cells upon treatment with
Cytometric assessment of CD133− and CD133+ and their cell cycle in
Sodium valproate (VPA) interactions with DNA and histones - ScienceDirect
Sodium valproate (VPA) interactions with DNA and histones - ScienceDirect
DNA demethylation by 5-aza-2′-deoxycytidine is imprinted, targeted to euchromatin, and has limited transcriptional consequences, Epigenetics & Chromatin
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